regular is shortĪc琀椀ng, 30 min to 1 hr, peak is 1-5 hrs, and dura琀椀on 6-10 hrs. Nph is an intermediate ac琀椀ng- 1-2 hrs onset, peak 6-14 hrs, and 16-24 dura琀椀on. Repeat BG and retreat q15 un琀椀l BG is > 70 mg/dL. Of orange juice and then a rn can check a昀琀er 15 mins and give 4 more oz of orange juice if theyįirst d/c the insulin orders so the blood glucose levels do no decrease any further. If the pa琀椀ent is conscious then she can have 4 oz This pa琀椀ent is hypoglycemic due to symptoms. In which the injec琀椀on is going to be given.Īssess TC an 15-30 mins a昀琀er administra琀椀ng the insulin as this is when the onset will occur. Air should also be inserted into the vial with the sameĪmount which is being admi琀琀ed in order to create a vacuum. Before giving the coverage, the insulin top Lispro insulin 6 units subq would be the best choice. Makes the pa琀椀ent more suscep琀椀ble to foot injuries, and uclers. It is 琀椀ngling, sharp pain, or numbness of the hands and feet. DM paresthesia is peripheral pain that is described as These pa琀椀ents vision will become impaired which the RN will need to teach DM re琀椀nopathy is damage to the eyes from nerve and vesselĭamage of the eye. Having a decreased ability to feel sensa琀椀on which makes these individuals prone to infec琀椀onsįrom untreated cuts and bruises. Pa琀椀ent will not exhibit hypoglycemic symptoms due to a sudden drop.ĭM neuropathy is in which the disease has induced nerve damage. The purpose of the 150mL/hr of D5 ½ NS is to control drops in glucose and due to this the Regular insulin onset is 30 mins-1 hr, 2-1. Regular insulin is given in IV given to those in DKA. Is about 2 hrs, there is no peak, and the dura琀椀on is 24 hrs. Glargine is a long ac琀椀ng insulin while lispro is a rapid ac琀椀ng insulin. Fairly controlled is indicated by 8.5-9 and poorly controlled is A1c greater than 9.ĪDA diet is a diet for diabe琀椀c pa琀椀ent to minimize not only their glucose consump琀椀on but also Good control is indicated by a A1c ofħ.5-8. 昀椀nds the average blood sugar levels over the last 3 months. A decrease pH indicates a decrease in Serumīicarbonate and an increase in ketones which can lead to DKA. The most important lab values for DKA are glucose levels, arterial pH, bicarbonate levels, and The pa琀椀ent TC presented with DKA as her blood sugar upon As a result of the breakdown of fat ketones occur while with hypoglycemia the body does not have enough sugar or insulin. With DKA the body is not producing insulin which leads to where the body cannot break down sugar therefore our body starts to break down fat instead. Hypoglycemia can present with tachycardia, fatigue, polyphagia, shakiness, sweating, dizziness, paleness, visual disturbances, seizures, and anxiety. Also with DKA is the blood sugar exceeds normal limits versus hypoglycemia it is below normal limits. Speci昀椀c signs on examina琀椀on for DKA are Kussmaul breathing and fruity odor. Addi琀椀onal symptoms include increased thirst (polydipsia)Īnd increased urina琀椀on (polyuria). The increase of 昀氀uid loss with polyuria.ĭKA is a complica琀椀on of hyperglycemia. The reason for polydipsia is because the body needs 昀氀uids due to The reason for polyphagia is because the glucose from the bloodĬannot enter the cells due to the lack of insulin or insulin resistance therefore the body cannotĬonvert it into energy so the body becomes fa琀椀gue and creates excessive need forįood/substance in response. Glucose ends up in the urine and ends up pulling more water which results in abnormally The reason why polyuria occurs in those with DM is because excessive Polydipsia is excessive thirst and polyphagia Produces abnormally large volume of diluted urine. The three P’s of DM1 is polyuria, polydipsia, and polyphagia. To insulin but if all other treatments fail to control DM2 the pa琀椀ent will use insulin to control Treatment forĭM1 must include insulin versus DM2 is a combina琀椀on of diet, exercise, alterna琀椀ve medicine DM2 is when theīody produces glucose but the cells in the body can not respond to it properly. Is caused by an autoimmune response against insulin producing beta cells. The major di昀昀erence between DM1 and DM2 is that DM1 is a gene琀椀c disorder that shows upĮarly in life versus DM2 is diet related and occurs slowly over 琀椀me.
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